In cases of acute closure angle glaucoma, what would likely be observed regarding pupil response to light?

In acute angle closure glaucoma, the pupil typically presents as fixed and nonreactive to light. This phenomenon occurs due to a combination of factors related to the acute rise in intraocular pressure and the involvement of the ciliary body. When the angle between the iris and the cornea narrows or closes, it obstructs the outflow of aqueous humor, leading to a rapid increase in intraocular pressure. The increased pressure can cause reflexive paralysis of the iris sphincter or ciliary muscles, resulting in a fixed dilated pupil that does not respond appropriately to light stimuli.

In this scenario, the other options describe pupil responses that would not be consistent with acute angle closure glaucoma. For instance, brisk or excessively constricted reactions would suggest more typical responses seen in other forms of ocular pathology, such as in cases of a direct light response or pharmacological agents affecting pupil dynamics. A normal pupil response would also be inconsistent given the significant intraocular pressure changes characteristic of this condition. Thus, a fixed and nonreactive pupil is a hallmark of acute angle closure glaucoma.